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However, the increased demand for cholesterol can be satisfied by increased uptake of extracellular cholesterol from LDL and HDL via endocytosis mediated by the surface receptors LDLR and scavenger receptor class B, type I (SCARB1) [16]

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In patients unfortunate enough to have suffered multiple episodes, these abnormalities intensify, resulting in higher overall cortisol levels in addition to aberrant reactivity, and even greater flattening of their cortisol curves, compared with patients who have experienced only a few episodes (173)